Association of novel genetic Loci with circulating fibrinogen levels: a genome-wide association study in 6 population-based cohorts.

نویسندگان

  • Abbas Dehghan
  • Qiong Yang
  • Annette Peters
  • Saonli Basu
  • Joshua C Bis
  • Alicja R Rudnicka
  • Maryam Kavousi
  • Ming-Huei Chen
  • Jens Baumert
  • Gordon D O Lowe
  • Barbara McKnight
  • Weihong Tang
  • Moniek de Maat
  • Martin G Larson
  • Susana Eyhermendy
  • Wendy L McArdle
  • Thomas Lumley
  • James S Pankow
  • Albert Hofman
  • Joseph M Massaro
  • Fernando Rivadeneira
  • Melanie Kolz
  • Kent D Taylor
  • Cornelia M van Duijn
  • Sekar Kathiresan
  • Thomas Illig
  • Yurii S Aulchenko
  • Kelly A Volcik
  • Andrew D Johnson
  • Andre G Uitterlinden
  • Geoffrey H Tofler
  • Christian Gieger
  • Bruce M Psaty
  • David J Couper
  • Eric Boerwinkle
  • Wolfgang Koenig
  • Christopher J O'Donnell
  • Jacqueline C Witteman
  • David P Strachan
  • Nicholas L Smith
  • Aaron R Folsom
چکیده

BACKGROUND Fibrinogen is both central to blood coagulation and an acute-phase reactant. We aimed to identify common variants influencing circulation fibrinogen levels. METHODS AND RESULTS We conducted a genome-wide association analysis on 6 population-based studies, the Rotterdam Study, the Framingham Heart Study, the Cardiovascular Health Study, the Atherosclerosis Risk in Communities Study, the Monitoring of Trends and Determinants in Cardiovascular Disease/KORA Augsburg Study, and the British 1958 Birth Cohort Study, including 22 096 participants of European ancestry. Four loci were marked by 1 or more single-nucleotide polymorphisms that demonstrated genome-wide significance (P<5.0 x 10(-8)). These included a single-nucleotide polymorphism located in the fibrinogen beta chain (FGB) gene and 3 single-nucleotide polymorphisms representing newly identified loci. The high-signal single-nucleotide polymorphisms were rs1800789 in exon 7 of FGB (P=1.8 x 10(-30)), rs2522056 downstream from the interferon regulatory factor 1 (IRF1) gene (P=1.3 x 10(-15)), rs511154 within intron 1 of the propionyl coenzyme A carboxylase (PCCB) gene (P=5.9 x 10(-10)), and rs1539019 on the NLR family pyrin domain containing 3 isoforms (NLRP3) gene (P=1.04 x 10(-8)). CONCLUSIONS Our findings highlight biological pathways that may be important in regulation of inflammation underlying cardiovascular disease.

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عنوان ژورنال:
  • Circulation. Cardiovascular genetics

دوره 2 2  شماره 

صفحات  -

تاریخ انتشار 2009